Your Weight Jumps Around Daily

why your weight jumps around so much from day to day

why your weight jumps around so much from day to day

If you’ve ever gotten into the habit of weighing yourself every day, you’ll have noticed something a little strange about the numbers on your bathroom scales. They’re all over the place. From day to day, it almost like you’re weighing a different person. The numbers seem to yo-yo up and down irrespective of how much you eat, drink, or exercise.

And if you’re actively trying to lose weight it, it’s not just confusing – it can be downright disheartening too. Nothing kills diet motivation and willpower quite like seeing those numbers go up when all your hard work and snack-sacrificing means they ought to be going down.

But according to Martin Robbins at The Guardian, it makes perfect sense that the numbers on your bathroom scale don’t make any sense. Why? Because there’s simply way too many things going on in your body all the time for individual measurements taken at any particular moment to be at all meaningful when viewed in isolation.

“Weight measurements are like opinion polls – individual results don’t tell you anything because there’s just too much random noise, error and variation,” he says. “It’s only when you have a few dozen that you can start to reliably pick out a trend.”

To get a better sense of all the ups and downs occurring in his weight, Robbins set himself an ambitious task: over a three-day long weekend, he weighed himself every waking hour to see what his body was up to. He also accurately recorded the specific weight of everything he ate and drank over the period, and even weighed the urine he passed. “I estimated the, er, other stuff – I do have some dignity,” he says.

At the end of three and a half days (from 6pm on Friday night to 9am on Tuesday morning), Robbins ingested a whopping 14.86 kg of consumables, consisting of 3.58 kg of food and 11.28 kg of drink. While that might sound like a lot – and it is – it’s not like he was all-out gorging himself the entire time. At the end of his experiment, he’d actually lost 1.86 kg, meaning his body had disposed of some 16.72 kg over the course of the weekend.

“7.4 kg of that was accounted for by urine, and an estimated 1.8 kg by, well, crap, but that still leaves a whopping 7.52 kg of mass that just vanished into thin air,” he says. “Where did it go?”

In his analysis, Robbins points out that we’re losing weight all the time in ways we never think about – ways that have nothing to do with eating healthily or going to the gym. Admittedly, a couple of 5 km runs he took over the weekend saw him displace over a kilogram in sweat. But there were still several kilograms of unaccounted weight loss: 69 grams per hour (1.65 kg every 24 hours) that couldn’t be otherwise explained.

“In fact, I really was evaporating into thin air. Humans breathe in oxygen, and breathe out carbon dioxide – oxygen plus a carbon atom. All those carbon atoms have to come from somewhere, and they add up pretty quickly – over the course of a day, with a good work out thrown in, someone my size breathes out maybe half a kilo of carbon,” he says.

Robbins estimates that we lose about the same amount again in exhaled water vapour, and then yet again by leaking water from our skin. Once he added up the estimated losses from these sources over the weekend, the riddle of his mystery weight loss over the weekend was solved.

“It also reveals another surprising truth; that when it comes to ditching mass from your body the anus really does bring up the rear end,” Robbins says. “My penis, lungs and skin all managed to outperform my posterior when it came to taking out the trash.”

Robbins’s conclusion is that in light of all the things our bodies are doing that results in us losing weight, there’s little or no point getting hung up on a number you don’t like taken from one seemingly random weigh-in taken at one point during the day.

“None of this is massively surprising of course, but what I think it shows is just how unreliable any single measurement of weight is,” he says. “On any given day my weight varied by about four pounds [1.8 kg], with a dozen pounds [5.4 kg] passing in and out of the giant meat tube that is me at only vaguely predictable times. When you consider that a sensible weight loss target is maybe 0.25 lbs [110 grams] per day, you can see how on most days that’s just going to be swallowed up in the noise.”

The best way to weigh yourself then – especially if you’re the kind who’s going to be at all emotional about it, which is probably most of us – is to stand on the scales but not attribute any particular importance to the number you see. Instead, just jot it down and calculate long-term trends over time, as that’s the closest we can get to seeing how our bodily weight is really changing.

“Weigh yourself every morning, but ignore the number that comes up on the scales,” says Robbins. “Instead take the average of the last seven days (preferably ten or fourteen), and after several weeks look at how that average is changing over time. That’s where the real truth lies.”

 

Source:  Sciencealert.com

Antioxidant diet could extend life

Antioxidant

Antioxidant

University of Florida Health researchers have found that putting people on a feast-or-famine diet may mimic some of the benefits of fasting, and that adding antioxidant supplements may counteract those benefits.

Fasting has been shown in mice to extend lifespan and to improve age-related diseases. But fasting every day, which could entail skipping meals or simply reducing overall caloric intake, can be hard to maintain.

“People don’t want to just under-eat for their whole lives,” said Martin Wegman, an M.D.-Ph.D. student at the UF College of Medicine and co-author of the paper recently published in the journal Rejuvenation Research. “We started thinking about the concept of intermittent fasting.”

Michael Guo, a UF M.D.-Ph.D. student who is pursuing the Ph.D. portion of the program in genetics at Harvard Medical School, said the group measured the participants’ changes in weight, blood pressure, heart rate, glucose levels, cholesterol, markers of inflammation and genes involved in protective cell responses over 10 weeks.

“We found that intermittent fasting caused a slight increase to SIRT 3, a well-known gene that promotes longevity and is involved in protective cell responses,” Guo said.

The SIRT3 gene encodes a protein also called SIRT3. The protein SIRT3 belongs to a class of proteins called sirtuins. Sirtuins, if increased in mice, can extend their lifespans, Guo said. Researchers think proteins such as SIRT3 are activated by oxidative stress, which is triggered when there are more free radicals produced in the body than the body can neutralize with antioxidants. However, small levels of free radicals can be beneficial: When the body undergoes stress — which happens during fasting — small levels of oxidative stress can trigger protective pathways, Guo said.

“The hypothesis is that if the body is intermittently exposed to low levels of oxidative stress, it can build a better response to it,” Wegman said.

The researchers found that the intermittent fasting decreased insulin levels in the participants, which means the diet could have an anti-diabetic effect as well.

The group recruited 24 study participants in the double-blinded, randomized clinical trial. During a three-week period, the participants alternated one day of eating 25 percent of their daily caloric intake with one day of eating 175 percent of their daily caloric intake. For the average man’s diet, a male participant would have eaten 650 calories on the fasting days and 4,550 calories on the feasting days. To test antioxidant supplements, the participants repeated the diet but also included vitamin C and vitamin E.

At the end of the three weeks, the researchers tested the same health parameters. They found that the beneficial sirtuin proteins such as SIRT 3 and another, SIRT1, tended to increase as a result of the diet. However, when antioxidants were supplemented on top of the diet, some of these increases disappeared. This is in line with some research that indicates flooding the system with supplemental antioxidants may counteract the effects of fasting or exercise, said Christiaan Leeuwenburgh, Ph.D., co-author of the paper and chief of the division of biology of aging in the department of aging and geriatric research.

“You need some pain, some inflammation, some oxidative stress for some regeneration or repair,” Leeuwenburgh said. “These young investigators were intrigued by the question of whether some antioxidants could blunt the healthy effects of normal fasting.”

On the study participants’ fasting days, they ate foods such as roast beef and gravy, mashed potatoes, Oreo cookies and orange sherbet — but they ate only one meal. On the feasting days, the participants ate bagels with cream cheese, oatmeal sweetened with honey and raisins, turkey sandwiches, apple sauce, spaghetti with chicken, yogurt and soda — and lemon pound cake, Snickers bars and vanilla ice cream.

“Most of the participants found that fasting was easier than the feasting day, which was a little bit surprising to me,” Guo said. “On the feasting days, we had some trouble giving them enough calories.”

Leeuwenburgh said future studies should examine a larger cohort of participants and should include studying a larger number of genes in the participants as well as examining muscle and fat tissue.

Source:  eurekalert.org

Gluten Causes Weight Gain

The case against gluten seems to have been closed with recent research from a Brazilian research team that published a report in the January 2013 Journal of Nutritional Biochemistry. It seems to have put an exclamation point on the wheat belly controversy.

The Study


Lacking scientific data confirming the mechanics of how gluten may or may not affect obesity, the study was set up to examine the differences in specific genetic and biochemical markers between rats fed gluten and rats that were kept gluten free.

The “wheat belly” syndrome and how it leads to other health issues was the purpose of their research. The research team chose biological markers that could indicate the onset of obesity and metabolic syndrome, precursors to diabetes and cardiac issues.

Both groups of rats were fed high fat diets. But one group was gluten free and the other group’s diet was 4.5 percent gluten. Even without tracing their predetermined markers, it was obvious the gluten free mice exhibited weight loss without any trace of lipid (fat) excretion.

An Analysis Of The Study


Sayer Ji of GreenmedInfo.com proposed this analysis: “… the weight gain associated with wheat consumption has little to do with caloric content per se; rather, the gluten proteins … disrupt endocrine and exocrine processes within the body, as well as directly modulating nuclear gene expression … to alter mamalian metabolism in the direction of weight gain.”

This study report, according to Sayer Ji proves that the major factor of obesity is gluten, not calories. Considering that both groups of mice were fed high fat diets and the gluten free mice lost weight without excreting lipids also implies that fat free diets for losing weight are bogus. This has been suspected by other nutritional experts who’ve abandoned matrix thinking.

Sayer Ji recommends that those who are overweight, pre-diabetic, experiencing metabolic syndrome, or suffering from irritable bowel syndrome try avoiding gluten grains, especially wheat, to determine from experience if gluten is the underlying cause.

There is evidence that gluten can be a factor in gut and psychology syndrome (GAPS) and even autism. (http://www.naturalnews.com/033094_gut_health_brain.html)

So How Did Wheat, “The Staff of Life,” Become A Weed of Disease?


Wheat is not the same today. It has been agriculturally hybrid, not genetically lab engineered over some decades to resist fungus, grow more quickly, and be more pliable for industrial bread baking. As a consequence, 50-60 years ago wheat containing only five percent gluten has become 50 percent gluten today.

Agricultural resources used the hybrid process for wheat to accommodate the baking industry’s mechanical requirements of pliable proteins, leading to the 10-fold increase of wheat’s gluten.

The processed food industry’s concern for production efficiency and perception of consumer demands has focused on the bottom line with the usual disregard to negative health consequences.

Slightly different high speed methods of baking evolved over time. By artificially bleaching flour and adding “improvers” with often toxic additives and mixing the dough violently, loaves of bread could be baked, cooled, and packaged within a few, short hours. Cheap, unhealthy foods for many with massive profits for a few.

This is beginning to change with measures that seem to offset gluten’s damage for some. For example, Whole Foods has their own bakery providing fresh breads daily without bromides, which can displace the thyroid gland’s iodine contents and create hypothyroidism.

Other local bakeries may provide sprouted grain and real sourdough breads, which even some celiac sufferers manage to consume without adverse reactions.

 

Source:  hungryforchange.tv

Protein Drug mimic’s food deprivation

A Drug That Can Extend Life as Effectively As Dieting:

A Drug That Can Extend Life as Effectively As Dieting

A Drug That Can Extend Life as Effectively As Dieting

Many studies have shown that rigorous caloric restriction, or strict dieting, can increase longevity dramatically in lifeforms from yeast to humans. But a study released today shows one way to mimic the life-extending effects of food deprivation – using drugs.

A team of researchers in the UK explored the role of a protein known as S6K1, which turns out to play an extraordinary role in aging and age-related disease. When the researchers grew mice lacking the gene to produce S6K1, their mice lived significantly longer (see chart – the red lines are mice without S6K1). They also developed fewer age-related debilitating conditions.

A Drug That Can Extend Life as Effectively As Dieting

Female mice without S6K1 lived slightly longer than their male counterparts, and over 160 days longer than the control group. That means the female mouse lifespan increased by twenty percent.

Mice without S6K1 also lost weight, even if they ate more than ordinary mice. In other words, a substance that could block the expression of S6K1 would trick the body into thinking that you’d gone on a very rigorous diet. And it would make you healthier into an older age. The best part?

In their paper, the researchers conclude:

It might be possible to develop drug treatments that manipulate S6K1 and AMPK to achieve improved overall health in later life. Indeed, short-term rapamycin treatment reduces adiposity in mice, and metformin treatment [often used against type 2 diabetes] extends lifespan in short-lived mice.

This is good news, because often when researchers make discoveries related to longevity there is no immediate pathway to manufacturing a life-extending drug. For all of us who want to stay healthy in old age while still eating sugar and fat once in a while, let’s hope this research team starts testing a drug based on their S6K1 discovery – and soon.